A síndrome de Legg-Calvé-Perthes ou doença de Perthes é uma doença degenerativa da articulação da anca (quadril em português brasileiro) em que ocorre. El absceso del psoas es una enfermedad infrecuente en niños. Puede tener una en pediatría es primario; sin embargo, en ocasiones, puede ser de origen secundario y médula espinal, enfermedad de. Legg-Calvé-Perthes, entre otros Download Citation on ResearchGate | Enfermedad de Legg Calvé Perthes (LCP) en varios aspectos de la enfermedad de Legg Calvé Perthes y la evolución de caderas de August · Anales espanoles de pediatria.
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Am J Hematol, 44pp. Am J Hematol, 48pp. Nature,pp. Enfermdad in blood coagulation factor V associated with resistance to activated protein C.
Síndrome de Legg-Calvé-Perthes – Wikipédia, a enciclopédia livre
Blood, 85pp. Si continua navegando, consideramos que acepta su uso. Conclusions Epidemiological and laboratory data from this group ofpediatric patients and from the control group do not supportthe hypothesis that a thrombogenesis defect couldbe the underlying cause of avascular necrosis of the hipjoint.
Anal Biochem,pp. Acta Med Scand,pp. Idiopathic osteonecrosis, hypofibrinolysis, high plasminogen activator inhibitor, high Lp aand therapy with stanozolol. Lancet,pp. xe
Enfermedad de Legg-Calve-Perthes
Determination of plasminogen activator and its fast inhibitor in plasma. It pertthes been suggested that the cause of Perthes diseasemay be intravascular thrombosis induced by a potentialcongenital hemostatic disorder leading to conditions ofthrombophilia or hyperfibrinolysis. Non traumatic osteonecrosis of the femoral head: Complete study of hemostasis with coagulation and antithrombinfactors as well as study of thrombophilia andfibrinolysis in these patients could determine the prevalenceof hemostasis and fibrinolysis in this group of patients.
Aguirre Canyadell aI. Special laboratory evaluation of coagulation.
Anticoagulant protein C pathway defective in majority of thromboembolic patients. N Engl J Med,pp. The second international anticardiolipin standardization workshop.
Association of idiopathic venous thromboembolism with single point-mutation at Arg perthed factor. Pediatr Res, 35pp. J Med,pp.
Epidemiological and laboratory data from this group ofpediatric patients and from the control group do not supportthe hypothesis that a thrombogenesis defect couldbe the underlying cause of avascular necrosis of the hipjoint. The Kinston antiphospholipid group.
Blood, 80pp. Clin Orthop,pp.
Thromb Haemost, 69pp. Familial hypofibrinolysis and venous thrombosis. Blood collection in strongly acid citrate anticoagulant used in a study of dietary influence of basal Tpa activity. Familial idiopathic oeteonecrosis mediated by familial hypofibrinolysis witn high levels of plasminogen activator inhibitor.
Activated protein C resistance as an additional risk factor for thrombosis in protein C deficient families. Eenfermedad improve our services and products, we use “cookies” own or third parties authorized to show advertising related to client preferences through the analyses of navigation customer behavior. Thromb Res, 11pp.
Background It has been suggested that the cause of Perthes diseasemay be intravascular thrombosis induced by a potentialcongenital hemostatic disorder leading to conditions ofthrombophilia or hyperfibrinolysis. Am J Clin Pathol, peduatriapp.
Clin Othop,pp. You can change the settings or obtain more information by clicking here. Clin Chem, 32pp. Perthes’ disease and the relevance of thrombophilia.
Enfermedad de Legg-Calvé-Perthes – Síntomas y causas – Mayo Clinic
The remaining patients were considered withinthe normal range when age was taken into account. Relation of altered hemostasis to etiology. Resistance to activated protein Perhes and Legg-Perthes disease. Br J Hematol, 71pp.